William J. Kaiser
William J. Kaiser
University of Texas Health Science Center
Verified email at uthscsa.edu
Cited by
Cited by
Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018
L Galluzzi, I Vitale, SA Aaronson, JM Abrams, D Adam, P Agostinis, ...
Cell Death & Differentiation 25 (3), 486-541, 2018
RIP3 mediates the embryonic lethality of caspase-8-deficient mice
WJ Kaiser, JW Upton, AB Long, D Livingston-Rosanoff, LP Daley-Bauer, ...
Nature 471 (7338), 368-372, 2011
Toll-like receptor 3-mediated necrosis via TRIF, RIP3, and MLKL
WJ Kaiser, H Sridharan, C Huang, P Mandal, JW Upton, PJ Gough, ...
Journal of Biological Chemistry 288 (43), 31268-31279, 2013
Virus inhibition of RIP3-dependent necrosis
JW Upton, WJ Kaiser, ES Mocarski
Cell host & microbe 7 (4), 302-313, 2010
DAI/ZBP1/DLM-1 complexes with RIP3 to mediate virus-induced programmed necrosis that is targeted by murine cytomegalovirus vIRA
JW Upton, WJ Kaiser, ES Mocarski
Cell host & microbe 11 (3), 290-297, 2012
RIP3 induces apoptosis independent of pronecrotic kinase activity
P Mandal, SB Berger, S Pillay, K Moriwaki, C Huang, H Guo, JD Lich, ...
Molecular cell 56 (4), 481-495, 2014
Apoptosis induced by the toll-like receptor adaptor TRIF is dependent on its receptor interacting protein homotypic interaction motif
WJ Kaiser, MK Offermann
The Journal of Immunology 174 (8), 4942-4952, 2005
Cutting Edge: RIP1 kinase activity is dispensable for normal development but is a key regulator of inflammation in SHARPIN-deficient mice
SB Berger, V Kasparcova, S Hoffman, B Swift, L Dare, M Schaeffer, ...
The Journal of Immunology 192 (12), 5476-5480, 2014
Viral infection and the evolution of caspase 8-regulated apoptotic and necrotic death pathways
ES Mocarski, JW Upton, WJ Kaiser
Nature Reviews Immunology 12 (2), 79-88, 2012
Inhibitor of Apoptosis Proteins Physically Interact with and Block Apoptosis Induced by Drosophila Proteins HID and GRIM
D Vucic, WJ Kaiser, LK Miller
Molecular and cellular biology 18 (6), 3300-3309, 1998
Cutting edge: FAS (CD95) mediates noncanonical IL-1β and IL-18 maturation via caspase-8 in an RIP3-independent manner
L Bossaller, PI Chiang, C Schmidt-Lauber, S Ganesan, WJ Kaiser, ...
The Journal of Immunology 189 (12), 5508-5512, 2012
Inhibition of reaper-induced apoptosis by interaction with inhibitor of apoptosis proteins (IAPs)
D Vucic, WJ Kaiser, AJ Harvey, LK Miller
Proceedings of the National Academy of Sciences 94 (19), 10183-10188, 1997
Caspase-8 and RIP kinases regulate bacteria-induced innate immune responses and cell death
D Weng, R Marty-Roix, S Ganesan, MK Proulx, GI Vladimer, WJ Kaiser, ...
Proceedings of the National Academy of Sciences 111 (20), 7391-7396, 2014
RIP1 suppresses innate immune necrotic as well as apoptotic cell death during mammalian parturition
WJ Kaiser, LP Daley-Bauer, RJ Thapa, P Mandal, SB Berger, C Huang, ...
Proceedings of the National Academy of Sciences 111 (21), 7753-7758, 2014
Receptor-interacting protein homotypic interaction motif-dependent control of NF-κB activation via the DNA-dependent activator of IFN regulatory factors
WJ Kaiser, JW Upton, ES Mocarski
The Journal of Immunology 181 (9), 6427-6434, 2008
RIPK3 activates parallel pathways of MLKL-driven necroptosis and FADD-mediated apoptosis to protect against influenza A virus
S Nogusa, RJ Thapa, CP Dillon, S Liedmann, TH Oguin III, JP Ingram, ...
Cell host & microbe 20 (1), 13-24, 2016
Divergent host responses during primary simian immunodeficiency virus SIVsm infection of natural sooty mangabey and nonnatural rhesus macaque hosts
G Silvestri, A Fedanov, S Germon, N Kozyr, WJ Kaiser, DA Garber, ...
Journal of virology 79 (7), 4043-4054, 2005
TNFR1-dependent cell death drives inflammation in Sharpin-deficient mice
JA Rickard, H Anderton, N Etemadi, U Nachbur, M Darding, N Peltzer, ...
Elife 3, e03464, 2014
Herpes simplex virus suppresses necroptosis in human cells
H Guo, S Omoto, PA Harris, JN Finger, J Bertin, PJ Gough, WJ Kaiser, ...
Cell host & microbe 17 (2), 243-251, 2015
Cytomegalovirus M45 cell death suppression requires receptor-interacting protein (RIP) homotypic interaction motif (RHIM)-dependent interaction with RIP1
JW Upton, WJ Kaiser, ES Mocarski
Journal of Biological Chemistry 283 (25), 16966-16970, 2008
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